Burning Questions

What makes for a “Hyperinflammatory Immune Synapse”?

What’s the IL-18 receptor doing on granulocytes?

Where’s all that IL-18 coming from?

 

How can IL-18 blockade improve the lives of patients with auto- and hyperinflammatory disease.

Why don’t patients with PSTPIP1 mutations get MAS?

By what mechanisms does IL-18 promote auto- and hyperinflammatory diseases?

 
 
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